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    More Than 800+ Bonemarrow Transplant

    Team with more than 800 Bonemarrow Transplant Under Its Belt

    High Success Rate For Haploidentical Transplant(Half Match Transplant) With Over 10 Years of Exprience.

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    80% Success rate

    More Than 80% Success rate For BonrMarrow Transplant.We Have All Solution At One Place In Blood Cancer.

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    100% Donor Success

    We Have 100% Donor Success Record for Donor

    We are one of the most Trusted Brand name in the field of Bone Marrow Transplant in India

    In Affordalbe Price

Bonemarrow Transplant

Opening Hours
Mon – Wed - 9:00 AM - 7:00 PM
Thursday - 9:00 AM - 6:30 PM
Saturday - 9:00 AM - 6:00 PM
Sunday - CLOSED

Dr. Vikas Dua

Head Of Department, PHO
Institute of Excellence in Bonemarrow Transplant

Dr. Vikas Dua is Paediatric Hemato Oncologist and Bone Marrow Transplant specialist. Dr. Dua’s results in the field of Paediatric Haematology Oncology and BMT have been some of the best. He and his team has donemore than 800+ Paediatric transplants. He is known for his excellent outcome in stem cell transplant especially in Pediatric Haploidentical Transplant and he has done some of the very rare transplants which nobody else has done in India...Read More

Emergency Cases

+91 9643750957

Emergency Helpline number for Prompt Response. You can Reach us on Whatsapp/ Viber / Emo Or Telegram +91 9643750957

Qualified Doctors

Our team of dedicated doctors is highly qualified and committed to excellence in providing an aesthetically pleasing environment to ensure quality in medical care.

Modern Equipment

Global standard of infection control practices and Innovative immunotherapies to cure blood cancers with State of Art facilities.

Emergency Help

We Are Available 24/7 For Emergency Cases,So No Worries In Case Of Emergency contact us on +91 9643750957

Individual Approach

Personalised care for each patient as its about Life, and we understand each one of you are important to us.

Types Of Blood Disorders

Followings are the Types Of Blood Disorder Commonly Found Which Can Be Cured By Bonemarrow Transplant.

What Our Patients Say

About Institute of Excellence in Bonemarrow Transplant

Clinic with Innovative Approach to Treatment

Our motive is to provide Best Service to patients, because we value each life and understand the importance of each life.


Years Expriences In Full And Half Match Transplant.


Countries Patients


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Thalassemia is an inherited blood disorder characterised by less oxygen-carrying protein (haemoglobin) and fewer red blood cells in the body than normal. Symptoms include fatigue, weakness, paleness and slow growth.

Mild forms may not need treatment.

Severe forms may require blood transfusions or a donor stem-cell transplant.

People with thalassemia may have mild or severe anemia

Because thalassemias are inherited, the condition sometimes runs in families

The disorder results in large numbers of red blood cells being destroyed, which leads to anemia.

Traits for thalassemia are more common in people from Mediterranean countries, like Greece and Turkey, and in people from Asia, Africa, and the Middle East

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Aplastic Anaemia

Aplastic anemia is an autoimmune disease in which the body fails to produce blood cells in sufficient numbers. Blood cells are produced in the bone marrow by stem cells that reside there. Aplastic anaemia causes a deficiency of all blood cell types.

Symptoms may develop slowly or suddenly. Fatigue, frequent infections, rapid heart rate and bleeding may occur.

It is more frequent in people in their teens and twenties, but is also common among the elderly

Aplastic anemia can be caused by exposure to certain chemicals, drugs, radiation, infection, immune disease

Aplastic anemia is present in up to 2% of patients with acute viral hepatitis.

More frequently parvovirus B19 is associated with aplastic crisis which involves only the red blood cells (despite the name).

Aplastic anemia involves all different cell lines.

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Immunodeficiency is a state in which the immune system's ability to fight infectious disease and cancer is compromised or entirely absent. Most cases of immunodeficiency are acquired ("secondary") due to extrinsic factors that affect the patient's immune system.

The cause of immunodeficiency varies depending on the nature of the disorder.

The cause can be either genetic or acquired by malnutrition and poor sanitary conditions.

Although there is no true discrimination to who this disease affects, the genes are passed from mother to child, and on occasion from father to child.

Available treatment falls into two modalities: treating infections and boosting the immune system.

Some deficiencies cause early mortality (before age one), others with or even without treatment are lifelong conditions.

Newer stem cell transplant technologies may lead to gene based treatments of debilitating and fatal genetic immune deficiencies.

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Metabolic Disorders

A metabolic disorder can happen when abnormal chemical reactions in the body alter the normal metabolic process. It can also be defined as inherited single gene anomaly, most of which are autosomal recessive. Some of the symptoms that can occur with metabolic disorders are lethargy, weight loss, jaundice and seizures.

The symptoms expressed would vary with the type of metabolic disorder.

There are four categories of symptoms: acute symptoms, late-onset acute symptoms, progressive general symptoms and permanent symptoms

Inherited metabolic disorders are one cause of metabolic disorders, and occur when a defective gene causes an enzyme deficiency.

These diseases, of which there are many subtypes, are known as inborn errors of metabolism.

Metabolic diseases can also occur when the liver or pancreas do not function properly.

Metabolic disorders can be present at birth, and many can be identified by routine screening.

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Inherited Bone Marrow

The classical Inherited Bone Marrow Failure Syndromes (IBMFS) such as Fanconi anemia, Dyskeratosis Congenita, Shwachman-Diamond syndrome, and Diamond-Blackfan anemia are a heterogeneous group of disorders, all of which are characterized by impaired hematopoiesis, varying degrees of peripheral cytopenias and marrow hypoplasia and dysplasia. Many of these are associated with an increased risk of clonal dominance and evolution to myelodyplastic syndrome (MDS) and acute myeloid leukemia (AML).

The genes responsible for a subset of IBMFS have been identified and will be reviewed.

Gene discovery in IBMFS has been difficult in large part due to the phenotypic heterogeneity of these syndromes.

Accurate diagnosis of IBMFS inform patient care as it allows appropriate screening of siblings to avoid choosing an affected donor if marrow transplant is indicated and the selection of an appropriate transplant conditioning regiment to avoid undue toxicity.

Additionally, accurate diagnosis allows appropriate medical monitoring and early intervention to successfully treat disease-specific non-hematologic medical complications.

The most common complications are iron overload in transfused patients and syndrome-specific malignancies in untransplanted patients,

The 4 most frequent syndromes are Fanconi anemia, dyskeratosis congenita, Diamond Blackfan anemia, and Shwachman Diamond syndrome.

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Failure Syndrome Like Fsnconi's Anemia

Fanconi anemia is a rare but serious blood disorder that prevents your bone marrow from making enough new blood cells for your body to work normally. It can also cause your bone marrow, the sponge-like tissue inside your bones, to make abnormal blood cells.Fanconi anemia is an inherited disease caused by mutations in certain genes, known as FA genes.

Fanconi anemia can lead to serious complications such as bone marrow failure, which happens when the bone marrow stops making as many blood cells.

The most common test for Fanconi anemia is a blood test called a chromosomal breakage test.

Treatment for Fanconi anemia depends on your age and how well your bone marrow is making new blood cells.

FA is the result of a genetic defect in a cluster of proteins responsible for DNA repair via homologous recombination.

Among those affected, the majority develop cancer, most often acute myelogenous leukemia, and 90% develop bone marrow failure (the inability to produce blood cells) .

FA is characterized by bone marrow failure, AML, solid tumors, and developmental abnormalities.

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Leukemia's And Lymphoma's

Leukemia and lymphoma are two types of cancer that affect the blood. Both cancers typically affect the white blood cells.Leukemia occurs when the bone marrow produces too many abnormal white blood cells. It is typically a slow-growing cancer, though there are cases where it progresses faster.

If a person has leukemia, their abnormal white blood cells do not die off in a normal cycle. Instead, the white blood cells multiply rapidly.

Eventually leaving less room for red blood cells required to carry oxygen through the body.

There are four main types of leukemia, classified according to their rate of growth and where the cancer originated in the body.

The types of leukemia include: acute lymphocytic leukemia, chronic lymphocytic leukemia, acute myeloid leukemia And chronic myeloid leukemia

Lymphoma starts in the immune system and affects the lymph nodes and lymphocytes, which are a type of white blood cell.

Cancer can affect all parts of the body, including the blood. Leukemia and lymphoma are types of blood cancer.

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Sickel Cell Anemia

Sickle cell anemia is an inherited form of anemia — a condition in which there aren't enough healthy red blood cells to carry adequate oxygen throughout your body. Normally, your red blood cells are flexible and round, moving easily through your blood vessels. In sickle cell anemia, the red blood cells become rigid and sticky and are shaped like sickles or crescent moons.

These irregularly shaped cells can get stuck in small blood vessels, which can slow or block blood flow and oxygen to parts of the body.

Sickle cells break apart easily and die, leaving you without enough red blood cells.

Red blood cells usually live for about 120 days before they need to be replaced. But sickle cells usually die in 10 to 20 days, leaving a shortage of red blood cells (anemia).

Periodic episodes of pain, called crises, are a major symptom of sickle cell anemia.

The pain varies in intensity and can last for a few hours to a few weeks. Some people have only a few pain episodes.

Sickle cells can damage an organ that fights infection (spleen), leaving you more vulnerable to infections.

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